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pmid: 34915813
pmc: PMC8735834
Cas9 overexpression in 3T3-L1 cell line
Physiology, adipocytes, Technical Reports, adipocyte function, cas9, Diseases of the endocrine glands. Clinical endocrinology, Cell Line, 3t3-l1, Mice, 3T3-L1 Cells, Adipocytes, QP1-981, Animals, Humans, Adipogenesis, QH573-671, Cell Differentiation, RC648-665, crispr/cas, genetic knock-out, Adipocytes, Cas9, CRISPR/Cas, adipocyte function, 3T3-L1, genetic knock-out, Feasibility Studies, CRISPR-Cas Systems, Cytology
Physiology, adipocytes, Technical Reports, adipocyte function, cas9, Diseases of the endocrine glands. Clinical endocrinology, Cell Line, 3t3-l1, Mice, 3T3-L1 Cells, Adipocytes, QP1-981, Animals, Humans, Adipogenesis, QH573-671, Cell Differentiation, RC648-665, crispr/cas, genetic knock-out, Adipocytes, Cas9, CRISPR/Cas, adipocyte function, 3T3-L1, genetic knock-out, Feasibility Studies, CRISPR-Cas Systems, Cytology
| selected citations These citations are derived from selected sources. This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 5 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Top 10% | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Average | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Average |
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