
The immune disorder PLAID is found to be the result of destabilization of B cell receptor complexes due to loss of an autoinhibitory domain in phospholipase C–γ2.
Analysis of Variance, Microscopy, Confocal, Phospholipase C gamma, Lipid Bilayers, Immunologic Deficiency Syndromes, Intracellular Signaling Peptides and Proteins, Receptors, Antigen, B-Cell, Protein-Tyrosine Kinases, Flow Cytometry, Microscopy, Fluorescence, Multiprotein Complexes, COS Cells, Chlorocebus aethiops, Agammaglobulinaemia Tyrosine Kinase, Animals, Humans, Syk Kinase, Calcium, Phosphorylation, Signal Transduction
Analysis of Variance, Microscopy, Confocal, Phospholipase C gamma, Lipid Bilayers, Immunologic Deficiency Syndromes, Intracellular Signaling Peptides and Proteins, Receptors, Antigen, B-Cell, Protein-Tyrosine Kinases, Flow Cytometry, Microscopy, Fluorescence, Multiprotein Complexes, COS Cells, Chlorocebus aethiops, Agammaglobulinaemia Tyrosine Kinase, Animals, Humans, Syk Kinase, Calcium, Phosphorylation, Signal Transduction
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| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 10% | |
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