2,3-Diphosphoglycerate: A Physiological Inhibitor of Platelet Aggregation
Copyright policy )2,3-Diphosphoglycerate: A Physiological Inhibitor of Platelet Aggregation
2,3-Diphosphoglycerate (2,3-DPG) may inhibit the platelet release reaction and the irreversible aggregation of human blood platelets induced by adenosine diphosphate, epinephrine, or norepinephrine. The effects of 2,3-DPG on platelet aggregation were more pronounced in cases with low hematocrit (< 30 percent). Dipyridamole and vincaminor potentiated the antiaggregating effect of 2,3-DPG. Erythrocytes (10 3 to 10 4 per microliter) exhibited a similar antiaggregating effect, especially when secured from anemic patients.
Epinephrine, Platelet Aggregation, Drug Synergism, Dipyridamole, In Vitro Techniques, Diphosphoglyceric Acids, Adenosine Diphosphate, Norepinephrine, Platelet Adhesiveness, Hematocrit, Depression, Chemical, Humans, Blood Coagulation, Vinca Alkaloids
Epinephrine, Platelet Aggregation, Drug Synergism, Dipyridamole, In Vitro Techniques, Diphosphoglyceric Acids, Adenosine Diphosphate, Norepinephrine, Platelet Adhesiveness, Hematocrit, Depression, Chemical, Humans, Blood Coagulation, Vinca Alkaloids
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