
doi: 10.1113/eph8602181
pmid: 11429642
The contractility of the human uterus is under the fine control of a variety of interacting bioactive agents. During labour, the excitability of the uterus is drastically transformed in comparison with the non‐labour state and is manifest at the membrane level via the acivity of uterine ion channels. This article reviews the contribution of potassium (K+) channels to human uterine excitability.
Labor, Obstetric, Potassium Channels, 610, Electrophysiology, Uterine Contraction, Adenosine Triphosphate, Pregnancy, Myometrium, Humans, Calcium, Female, Ion Channel Gating
Labor, Obstetric, Potassium Channels, 610, Electrophysiology, Uterine Contraction, Adenosine Triphosphate, Pregnancy, Myometrium, Humans, Calcium, Female, Ion Channel Gating
| selected citations These citations are derived from selected sources. This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 95 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Top 10% | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 10% | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Top 10% |
