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{"references": ["1. Noh TW, Park SH, KangYS, Lee UH, Park HS, Jang SJ. Morphea developing at the site of healed herpes zoster. Ann Dermatol. 2011;23(2):242-245.", "2. Koley S, Saoji V, Salodkar A. Unusual formation of keloids after each episode of recurrent herpes zoster in an HIV positive patient. Indian J Sex Transm Dis. 2009;30(2): 109-111", "3. Ingordo V, Gentile C, Iannazzone SS, Cusano F, Naldi L. The 'EpiEnlist' project: a dermo epidemiologic study - on a representative sample of young Italian males. Prevalence of selected pigmentary lesions. J Eur Acad Dermatol Venereol, 2007;21(8):1091\u20136.", "4. Camacho D, Feltes F, Mach\u00e1n S, Pielasinski \u00da, Fari\u00f1a MC, Gavin E, Requena L. Primary cutaneous dermal mucinosis on herpes zoster scars. Cutis. 2016;98(1): E19-E23.", "5. Hafner C1, van Oers JM, Vogt T, Landthaler M, Stoehr R, Blaszyk H, etal. Mosaicismofactivating FGFR3 mutationsin human skin causes epidermal nevi. J Clin Invest 2006;116(8):2201\u20137."]}
Herpes zoster lesions are well known to heal with abnormal scar formation. Collagen deposition occurs as a result of abnormal immunological and inflammatory response within the tissue. Whereas, connective tissue nevi are formed by structural alteration or increase in collagen or elastin fibers, we report a case of a young male whose herpes zoster lesions healed with formation of a collagen nevus.
isotropic response, connective tissue nevi, herpes zoster
isotropic response, connective tissue nevi, herpes zoster
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