
Dilated Cardiomyopathy (DCM) is a disease of heart muscle manifested by enlargement and dilation of one or both of the ventricles along with impaired contractility narrated as left ventricular ejection fraction (LVEF) less than 40%. Patients may or may not have overt signs of heart failure, but they are by definition exhibiting systolic dysfunction. There are two types of DCM for this disease process: primary and secondary. Since primary DCM is thought to be idiopathic, secondary causes must be ruled out before the diagnosis can be made. This exercise describes the causes, symptoms, and importance of the interprofessional team in managing dilated cardiomyopathy.
Dilated Cardiomyopathy left ventricular ejection fraction, ventricular arrhythmias, thrombo embolism, desmin, lamin, myosin, myocarditis, lyme disease, chagas disease, balloning of the left venricular apex, catecholamine surge, neuro humoral activation, dyspnea, orthopenia, thrombo embolic complications, arrhythmias, tricuspid or mitral regurgatation murmur, hemo chromatosis and serum B-type natri uretic peptide
Dilated Cardiomyopathy left ventricular ejection fraction, ventricular arrhythmias, thrombo embolism, desmin, lamin, myosin, myocarditis, lyme disease, chagas disease, balloning of the left venricular apex, catecholamine surge, neuro humoral activation, dyspnea, orthopenia, thrombo embolic complications, arrhythmias, tricuspid or mitral regurgatation murmur, hemo chromatosis and serum B-type natri uretic peptide
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