
The development of myocardial infarction entails the occurrence of systemic and local inflammatory reactions, activation of acute-phase proteins, in particular — components of the complement system, C-reactive protein, orosomucoid, 1-antitrypsin, kallikrein, kinins [2]. Systemic modulation manifests itself in the development of fever, pain, leukocytosis, bone marrow stimulation, the appearance of prostaglandins, interferon, acute phase proteins, antibodies [3].
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