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A STUbL wards off telomere fusions

descriptionPublicationkeyboard_double_arrow_right Article 22 Feb 2013Publisher:Springer Science and Business Media LLCJournal:The EMBO Journal, volume 32, pages 775-777 (issn: 0261-4189, eissn: 1460-2075, Copyright policy )Funded by:NIH | Studies of the Smc5/Smc6 ...
Authors: Inn Chung; Xiaolan Zhao;

doi: 10.1038/emboj.2013.39

pmid: 23435563

pmc: PMC3604727

A STUbL wards off telomere fusions

Abstract

Protection of chromosomal ends from erroneous recognition as double-strand breaks (DSBs) is critical for maintaining genome integrity. Unprotected ends can activate DNA repair pathways leading to catastrophic fusions or rearrangements of chromosomes. A study in The EMBO Journal identifies Uls1 as a new player in this endprotection process, utilizing SUMO and ubiquitin modifications to prevent chromosomal fusions by clearing a polysumoylated form of Rap1.

Related Organizations
Keywords

DNA End-Joining Repair, Saccharomyces cerevisiae Proteins, DNA Helicases, Telomere

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citations
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
0
Average
Average
Average
gold