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Leishmania donovani Impedes Antileishmanial Immunity by Suppressing Dendritic Cells via the TIM-3 Receptor

descriptionPublicationkeyboard_double_arrow_right Article , Other literature type 30 Aug 2022 English Publisher:American Society for MicrobiologyJournal:mBio, volume 13 (eissn: 2150-7511, Copyright policy )Funded by:UKRI | A Global Network for Negl...
Authors: Md. Naushad Akhtar; Sahil Kumar; Pradip Sen;

doi: 10.1128/mbio.03309-21

pmid: 35924848

pmc: PMC9426438

Leishmania donovani Impedes Antileishmanial Immunity by Suppressing Dendritic Cells via the TIM-3 Receptor

Abstract

Visceral leishmaniasis (VL), a poverty-related disease caused by Leishmania donovani , is ranked by the World Health Organization as the second largest killer parasitic disease in the world. The protective immune response against VL is primarily regulated by dendritic cells (DCs), which upon activation/maturation initiate an antileishmanial immune response.

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Keywords

Mice, Inbred BALB C, immunoregulation, TIM-3, Antiprotozoal Agents, Mucins, Immunoglobulins, Dendritic Cells, Microbiology, NF-κB, QR1-502, Mice, visceral leishmaniasis, Animals, Humans, Leishmaniasis, Visceral, dendritic cells, Hepatitis A Virus Cellular Receptor 2, Research Article, Leishmania donovani

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    popularity
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    influence
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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
7
Top 10%
Average
Top 10%
Green
gold
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