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The Ephrin A1–EphA2 System Promotes Cardiac Stem Cell Migration After Infarction

descriptionPublicationkeyboard_double_arrow_right Article 29 Apr 2011 Italy English Publisher:Ovid Technologies (Wolters Kluwer Health)Journal:Circulation Research, volume 108, pages 1,071-1,083 (issn: 0009-7330, eissn: 1524-4571,

Authors: Goichberg P; Bai YN; D'Amario D; Ferreira-Martins J; Fiorini C; Zheng HQ; Signore S; +9 Authors

doi: 10.1161/circresaha.110.239459

pmid: 21415392

pmc: PMC3102645

handle: 10281/22068 , 11579/147201 , 11579/147144

The Ephrin A1–EphA2 System Promotes Cardiac Stem Cell Migration After Infarction

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Abstract

RETRACTION This article has been retracted: Circ Res . 2018;123:e00. DOI: 10.1161/RES.0000000000000249.

Country

Italy

Related Organizations

Brigham and Women's Faulkner Hospital
United States
University of Eastern Piedmont Amadeo Avogadro
Italy
Beth Israel Deaconess Medical Center
United States
Albert Einstein College of Medicine
United States
University of Milan
Italy

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Keywords

570, Cytoplasm, Cell Membrane, Green Fluorescent Proteins, Myocardial Infarction, Ephrin-A2, Gene Expression, Ephrin-A1, Mice, Transgenic, Hematopoietic Stem Cells, Rats, Inbred F344, Rats, Mice, Inbred C57BL, Mice, Cell Movement, Cell Adhesion, Tachycardia, Ventricular, Animals, Humans, Myocytes, Cardiac, c-kit/GFP transgene,cell migartion

5 Research products, page 1 of 1

cDNA FLJ75959, highly similar to Homo sapiens ephrin-A1 (EFNA1), transcript variant 2, mRNA
2017IsRelatedTo
Ephrin-A1
2017IsRelatedTo
Ephrin-A2
2017IsRelatedTo
Ephrin-A2 [amino acids 176-213]
2017IsRelatedTo
Uncharacterized protein
2017IsRelatedTo

Impact byBIP!

	citations This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).	61
	popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.	Top 10%
	influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).	Top 10%
	impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.	Top 1%