
Ligand-independent Notch signaling promotes blood cell survival during normal development and under hypoxic stress.
Hemocytes, Receptors, Notch, Cell Survival, Aryl Hydrocarbon Receptor Nuclear Translocator, Calcium-Binding Proteins, Cytoplasmic Vesicles, Membrane Proteins, Hypoxia-Inducible Factor 1, alpha Subunit, Ligands, Cell Hypoxia, Endocytosis, Hematopoiesis, DNA-Binding Proteins, Animals, Drosophila Proteins, Intercellular Signaling Peptides and Proteins, Drosophila, Serrate-Jagged Proteins, Jagged-1 Protein, Signal Transduction
Hemocytes, Receptors, Notch, Cell Survival, Aryl Hydrocarbon Receptor Nuclear Translocator, Calcium-Binding Proteins, Cytoplasmic Vesicles, Membrane Proteins, Hypoxia-Inducible Factor 1, alpha Subunit, Ligands, Cell Hypoxia, Endocytosis, Hematopoiesis, DNA-Binding Proteins, Animals, Drosophila Proteins, Intercellular Signaling Peptides and Proteins, Drosophila, Serrate-Jagged Proteins, Jagged-1 Protein, Signal Transduction
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