
Hepatopulmonary syndrome (HPS) affects 10%–30% of patients with cirrhosis and portal hypertension and significantly increases mortality. Studies in experimental models indicate that pulmonary angiogenesis contributes to the development of HPS, but pathogenesis in humans is poorly understood. We investigated genetic risk factors for HPS in patients with advanced liver disease.
Adult, Liver Cirrhosis, Male, Genetic Variation, Neovascularization, Physiologic, Middle Aged, Polymorphism, Single Nucleotide, Haplotypes, Risk Factors, Case-Control Studies, Humans, Female, Aged, Hepatopulmonary Syndrome
Adult, Liver Cirrhosis, Male, Genetic Variation, Neovascularization, Physiologic, Middle Aged, Polymorphism, Single Nucleotide, Haplotypes, Risk Factors, Case-Control Studies, Humans, Female, Aged, Hepatopulmonary Syndrome
| selected citations These citations are derived from selected sources. This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 73 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Top 10% | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 10% | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Top 10% |
