Sulfated oxysterol 25HC3S as a therapeutic target of non-alcoholic fatty liver disease

descriptionPublicationkeyboard_double_arrow_right Article , Other literature type 01 Aug 2012 English Publisher:Elsevier BVJournal:Metabolism, volume 61, pages 1,055-1,057 (issn: 0026-0495,

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Authors: Ji-Young, Cha; Young-Bum, Kim;

doi: 10.1016/j.metabol.2012.04.011

pmid: 22592130

Sulfated oxysterol 25HC3S as a therapeutic target of non-alcoholic fatty liver disease

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Beth Israel Deaconess Medical Center
United States
Gachon University
Korea (Republic of)
Charles R. Drew University of Medicine and Science
United States

Keywords

Male, Lipid Metabolism, Orphan Nuclear Receptors, Lipids, Fatty Liver, Liver, Animals, Female, Sulfotransferases, Sterol Regulatory Element Binding Protein 1, Signal Transduction

12 Research products, page 1 of 2

25-Hydroxycholesterol 3-Sulfate Recovers Acetaminophen Induced Acute Liver Injury via Stabilizing Mitochondria in Mouse Models
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Sulfated oxysterol, 25HC3S, is a potent regulator of lipid metabolism in human hepatocytes
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Cholesterol metabolites alleviate injured liver function and decrease mortality in an LPS-induced mouse model
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Sulfation of 25-hydroxycholesterol regulates lipid metabolism, inflammatory responses, and cell proliferation
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Oxysterol sulfation by cytosolic sulfotransferase suppresses liver X receptor/sterol regulatory element binding protein–1c signaling pathway and reduces serum and hepatic lipids in mouse models of nonalcoholic fatty liver disease
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Cholesterol metabolite, 5-cholesten-3β-25-diol-3-sulfate, promotes hepatic proliferation in mice
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Regulation of Hepatocyte Lipid Metabolism and Inflammatory Response by 25‐Hydroxycholesterol and 25‐Hydroxycholesterol‐3‐sulfate
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25-Hydroxycholesterol-3-sulfate regulates macrophage lipid metabolism via the LXR/SREBP-1 signaling pathway
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25-Hydroxycholesterol-3-sulfate attenuates inflammatory response via PPARγ signaling in human THP-1 macrophages
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Biosynthesis of the regulatory oxysterol, 5-cholesten-3β,25-diol 3-sulfate, in hepatocytes
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