PABPC4 Broadly Inhibits Coronavirus Replication by Degrading Nucleocapsid Protein through Selective Autophagy
Copyright policy )PABPC4 Broadly Inhibits Coronavirus Replication by Degrading Nucleocapsid Protein through Selective Autophagy
Emerging coronaviruses (CoVs) can cause severe diseases in humans and animals, but none of the currently available drugs or vaccines can effectively control these diseases. During viral infection, the host will activate the interferon (IFN) signaling pathways and host restriction factors in maintaining the innate antiviral responses and suppressing viral replication.
- Shanghai Veterinary Research Institute China (People's Republic of)
- Chinese Academy of Agricultural Sciences China (People's Republic of)
- Yangzhou University China (People's Republic of)
- Zhejiang Ocean University China (People's Republic of)
- Huazhong Agricultural University China (People's Republic of)
Sp1 Transcription Factor, Swine, coronaviruses, Ubiquitin-Protein Ligases, Infectious bronchitis virus, Microbiology, Poly(A)-Binding Proteins, N protein, Cell Line, Chlorocebus aethiops, PABPC4, Autophagy, Animals, Coronavirus Nucleocapsid Proteins, Humans, Vero Cells, selective autophagy, Murine hepatitis virus, Porcine epidemic diarrhea virus, Ubiquitination, Nuclear Proteins, Blood Proteins, QR1-502, SP1, Coronavirus, HEK293 Cells, Proteolysis, Research Article
Sp1 Transcription Factor, Swine, coronaviruses, Ubiquitin-Protein Ligases, Infectious bronchitis virus, Microbiology, Poly(A)-Binding Proteins, N protein, Cell Line, Chlorocebus aethiops, PABPC4, Autophagy, Animals, Coronavirus Nucleocapsid Proteins, Humans, Vero Cells, selective autophagy, Murine hepatitis virus, Porcine epidemic diarrhea virus, Ubiquitination, Nuclear Proteins, Blood Proteins, QR1-502, SP1, Coronavirus, HEK293 Cells, Proteolysis, Research Article
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