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Other literature type . 2021
License: CC BY
Data sources: Datacite
image/svg+xml art designer at PLoS, modified by Wikipedia users Nina, Beao, JakobVoss, and AnonMoos Open Access logo, converted into svg, designed by PLoS. This version with transparent background. http://commons.wikimedia.org/wiki/File:Open_Access_logo_PLoS_white.svg art designer at PLoS, modified by Wikipedia users Nina, Beao, JakobVoss, and AnonMoos http://www.plos.org/
figshare
Other literature type . 2021
License: CC BY
Data sources: Datacite
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Additional file 2 of PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

descriptionPublicationkeyboard_double_arrow_right Other literature type 01 Jan 2021Publisher:figshare
Authors: Yao, Bing; Gui, Tao; Zeng, Xiangwei; Deng, Yexuan; Wang, Zhi; Wang, Ying; Yang, Dongjun; +16 Authors

doi: 10.6084/m9.figshare.14423346 , 10.6084/m9.figshare.14423346.v1

Additional file 2 of PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

Abstract

Additional file 2: Supplementary tables. Table S1. Clinicopathologic characteristics of PRMT1 and SMARCA4 expression in CRC patients. Table S2. 15 common target genes listed both in ChIP-Seq and microarray data. Table S3. A list of antibodies used in the present study. Table S4. Peptides used in the present study. Table S5. The list of primer sequences for RT-PCR. Table S6. The list of primer sequences for ChIP. Table S7. The list of viruses used in the present study. Table S8: Tissue array information. Table S9. List of proteins identified by Mass Spectrometry. Table S10. 109 common genes down-regualted by PRMT1-knockdown and SMARCA4-knockdown in HCT116 cells.

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
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