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Conference object . 2025
License: CC BY
Data sources: ZENODO
ZENODO
Article . 2025
License: CC BY
Data sources: Datacite
ZENODO
Article . 2025
License: CC BY
Data sources: Datacite
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PARKINSON KASALLIGINING MOLEKULYAR MEXANIZMLARI

descriptionPublicationkeyboard_double_arrow_right Article , Conference object 10 Mar 2025Publisher:Fast support and result LLC
Authors: Uralova Muxlisa Ro'ziboy qizi, Turdimurodova Shahzoda Sarvar qizi;

doi: 10.5281/zenodo.15152930 , 10.5281/zenodo.15152929

PARKINSON KASALLIGINING MOLEKULYAR MEXANIZMLARI

Abstract

Parkinson kasalligi markaziy asab tizimining degenerativ kasalliklaridan biri bo‘lib, dopamin neyronlarining progressiv yo‘qolishi bilan xarakterlanadi. Parkinson kasalligi rivojlanishida genetika va atrof-muhit omillari muhim rol o‘ynaydi. Ushbu kasallikni davolashida hozirgi kunda dopaminergik terapiya asosiy usul sifatida qo‘llaniladi. Shuningdek, neyroprotektiv dorilar va gen terapiya kabi yangi yondashuvlar ham rivojlanmoqda. Kelajakda molekulyar mexanizmlarni chuqur o‘rganish orqali shaxsiylashtirilgan terapiya va kasallikni kechiktirish yoki to‘xtatish yo‘llari ishlab chiqilishi mumkin.

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
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Average
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