
In the therapy of chronic myelogenous leukemia (CML), interferon (IFN) brought about a big progress resulting in the good hematologic as well as cytogenetic response and prolongation of survival. Recently, imatinib, the BCR/ABL antagonist, took over the first choice drug of the treatment CML. However, IFN still has the possibility to contribute the improvement of the therapy efficacy of CML.
Pyrimidines, Leukemia, Myelogenous, Chronic, BCR-ABL Positive, Benzamides, Cytarabine, Imatinib Mesylate, Humans, Interferon-alpha, Antineoplastic Agents, Drug Therapy, Combination, Philadelphia Chromosome, Genes, abl, Piperazines
Pyrimidines, Leukemia, Myelogenous, Chronic, BCR-ABL Positive, Benzamides, Cytarabine, Imatinib Mesylate, Humans, Interferon-alpha, Antineoplastic Agents, Drug Therapy, Combination, Philadelphia Chromosome, Genes, abl, Piperazines
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