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Recolector de Ciencia Abierta, RECOLECTA
Bachelor thesis . 2016
License: CC BY NC ND
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Recolector de Ciencia Abierta, RECOLECTA
Bachelor thesis . 2016
License: CC BY NC ND
image/svg+xml art designer at PLoS, modified by Wikipedia users Nina, Beao, JakobVoss, and AnonMoos Open Access logo, converted into svg, designed by PLoS. This version with transparent background. http://commons.wikimedia.org/wiki/File:Open_Access_logo_PLoS_white.svg art designer at PLoS, modified by Wikipedia users Nina, Beao, JakobVoss, and AnonMoos http://www.plos.org/
UCrea
Bachelor thesis . 2016
License: CC BY NC ND
Data sources: UCrea
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Alteraciones del sueño, obesidad y resistencia a la insulina

Authors: Casanueva García de Alvear, María Asunción;

Alteraciones del sueño, obesidad y resistencia a la insulina

Abstract

Numerosos parámetros metabólicos, desde niveles hormonales, a concentraciones de nutrientes en plasma, oscilan a lo largo del día en relación a los ciclos de luz-oscuridad, ingesta, y comportamiento. La regulación del metabolismo corporal (metabolismo lipídico, glucídico y proteico) y las hormonas que lo regulan, se correlaciona de una forma intensa con la relación establecida entre el sueño y la ritmicidad circadiana. Por tanto alteraciones del sueño conducirán a la disrupción de los procesos metabólicos, y ejes hormonales encargados de la regulación de la homeostasis interna. Individuos que poseen afectado el sueño, ya sea por disminución de la calidad o reducción del número de horas del mismo, presentan: un incremento de la ingesta, aumento de la sensibilidad a estímulos placenteros, posible disminución de la actividad física voluntaria, e incremento de factores de riesgo para el desarrollo de diabetes tipo ll como por ejemplo la resistencia a la insulina. Se puede establecer que la falta de sueño debe ser considerada como importante factor de riesgo para el desarrollo de la obesidad y la diabetes tipo ll. Es por ello que el sueño debe ser establecido como otra medida para la promoción, prevención y tratamiento de la obesidad y la diabetes tipo ll.

Several metabolic parameters ranging from hormone levels and nutrient concentrations in plasma, fluctuate in time-of-the-day dependent manner and are associated to human behaviour and eating. Metabolic regulation (hormone axis, and lipid, carbohydrates, and protein and metabolism) is correlated in a very powerful way, with the relationship build up between sleep, circadian rhythmicity. As a consequence of that, inadequate sleep, in both quality or quantity, will enhance the impairment of metabolic processes and hormonal axis in charge of the internal homeostasis regulation. Individuals that suffer sleep disturbances present: an increased food intake, higher susceptibility to food stimuli, a posible reduction of voluntary physic activity, and an increased presence of certain predictors of type ll diabetes such as insulin resistance. In summary sleep disturbances should be regarded as an important risk factor for the development of obesity and type ll diabetes. In such a way that, a good night’s sleep should be egarded as a critical component in the promotion, prevention, and treatment of obesity and type ll diabetes.

Grado en Medicina

Country
Spain
Related Organizations
Keywords

Metabolismo, Metabolism, Diabetes, Circadian rhythmicity, Obesidad, Ritmos circadianos, Obesity, Sleep, Sueño

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selected citations
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This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
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popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
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impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
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