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p38δ and PKD1: Kinase Switches for Insulin Secretion

descriptionPublicationkeyboard_double_arrow_right Article 01 Jan 2009 Spain English Publisher:Elsevier BVJournal:Cell, volume 136, pages 209-210 (issn: 0092-8674, Copyright policy )
Authors: Cuenda, Ana; Nebreda, Ángel R.;

doi: 10.1016/j.cell.2009.01.005

pmid: 19167323

handle: 10261/11439

p38δ and PKD1: Kinase Switches for Insulin Secretion

Abstract

Insufficient production of the hormone insulin by pancreatic beta cells results in diabetes. In this issue, Sumara et al. (2009) report key roles for the protein kinases p38delta and PKD1 in the regulation of insulin secretion as well as in the survival of pancreatic beta cells.

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Spain
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Keywords

Mice, Mitogen-Activated Protein Kinase 13, Diabetes Mellitus, Type 2, Biochemistry, Genetics and Molecular Biology(all), Insulin secretion, Insulin-Secreting Cells, PKD1, Insulin Secretion, Animals, Insulin, Pancreatic beta cells, p38delta, Protein Kinase C

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
views
OpenAIRE UsageCountsViews provided by UsageCounts
137
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35
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