
A Drosophila model for a neurological disorder called type 2B Charcot-Marie-Tooth disease reveals that it has its origins in a partial loss of function, rather than a gain of function, which points to the need for a new therapeutic approach.
570, QH301-705.5, Science, synapse, Charcot-Marie-Tooth Disease, Animals, genetics, Biology (General), endosome, Q, Laminopathies, animal models of disease, R, rab7 GTP-Binding Proteins, Neurodegenerative Diseases, rab GTP-Binding Proteins, Mutation, Medicine, neuropathy, genetic, Neuroscience
570, QH301-705.5, Science, synapse, Charcot-Marie-Tooth Disease, Animals, genetics, Biology (General), endosome, Q, Laminopathies, animal models of disease, R, rab7 GTP-Binding Proteins, Neurodegenerative Diseases, rab GTP-Binding Proteins, Mutation, Medicine, neuropathy, genetic, Neuroscience
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