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Nihon Toseki Igakkai Zasshi
Article . 1995 . Peer-reviewed
Full-Text: http://www.jstage.jst.go.jp/article/jsdt1994/28/3/28_3_261/_pdf
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Nihon Toseki Igakkai Zasshi
Article . 1995 . Peer-reviewed
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Nihon Toseki Igakkai Zasshi
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https://dx.doi.org/10.4009/jsd...
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Four chronic hemodialysis patients whose intact-PTH level markedly decreased after administration of saccharated iron.

含糖酸化鉄剤の投与により血中副甲状腺ホルモンの低下を認めた血液透析患者の４例
descriptionPublicationkeyboard_double_arrow_right Article 01 Jan 1995Publisher:Japanese Society for Dialysis TherapyJournal:Nihon Toseki Igakkai Zasshi, volume 28, pages 261-265 (issn: 1340-3451, eissn: 1883-082X, Copyright policy )
Authors: Shibata, Masanori; Uchiyama, Hideo; Fukushima, Mitsuo; Taniguchi, Shinkichi; Usami, Kazumasa;

doi: 10.4009/jsdt.28.261

Four chronic hemodialysis patients whose intact-PTH level markedly decreased after administration of saccharated iron.

Abstract

鉄欠乏状態にあり, 貧血治療の目的で静注鉄剤を投与したところ, 血中intact-PTHレベルの顕著な低下を認めた4例を経験したので報告する. いずれも透析歴は4-9年と長期の維持透析を行っており, intact-PTHは145-1,050pg/mlと高く, うち2例では血清ALP活性, 血中Piともに高値であり, 二次性副甲状腺機能亢進症 (2°HPT) の状態と考えられた. 鉄剤投与によりintact-PTHは, 43-90%の低下を認め, 全例で投与中止後再上昇した. 進行した2°HPTの決定的内科的治療法のない現在, 鉄剤投与が今後一つの補助的方法として検討されるべきであろうと考えた.

Keywords

含糖酸化鉄剤, 二次性副甲状腺機能亢進症, 維持血液透析, 鉄欠乏状態, 2°HPT, intact-PTH

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
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