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German Medical Science
Conference object . 2014
License: CC BY NC ND
Data sources: German Medical Science
https://dx.doi.org/10.3205/14d...
Conference object . 2014
Data sources: Datacite
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Synoviocytes. Autonomous drivers of chronicity?

descriptionPublicationkeyboard_double_arrow_right Conference object 01 Jan 2014Embargo end date: 12 Sep 2014 English Publisher:German Medical Science GMS Publishing House; Düsseldorf
Authors: Böttcher, M; Schett, G; Falk, C; Irmler, I; Wunrau, C; Korb-Pap, A; Pap, T; +1 Authors

doi: 10.3205/14dgrh072

Synoviocytes. Autonomous drivers of chronicity?

Abstract

Background: Immunization with Glucose-6-phosphate isomerase (G6PI) induces arthritis in susceptible strains of mice. Depletion of regulatory T cells (Tregs) prior to immunization switches the usually acute, self-limiting course to a non-remitting, destructive arthritis. This provides a possibility to[for full text, please go to the a.m. URL]

42. Kongress der Deutschen Gesellschaft für Rheumatologie (DGRh); 28. Jahrestagung der Deutschen Gesellschaft für Orthopädische Rheumatologie (DGORh); 24. wissenschaftliche Jahrestagung der Gesellschaft für Kinder- und Jugendrheumatologie (GKJR)

Keywords

rheumatoid arthritis, ddc: 610, synovial fibroblasts, 610 Medical sciences; Medicine

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
0
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