<script type="text/javascript">
<!--
document.write('<div id="oa_widget"></div>');
document.write('<script type="text/javascript" src="https://www.openaire.eu/index.php?option=com_openaire&view=widget&format=raw&projectId=undefined&type=result"></script>');
-->
</script>
pmid: 13336070
Conclusions1. Total circulatory occlusion alone does not slow the rate of the normother-mic or hypothermic heart. 2. Intracoronary acetylcholine will profoundly decrease the cardiac rate in the occluded heart. The duration of this effect is short at normal body temperatures but quite prolonged during hypothermia (24–27°C). 3. Intracoronary atropine readily reverses the acetylcholine effect on the heart. 4. Cardiac slowing or arrest affords an added protection to the hypothermic heart during total circulatory occlusion.
Heart, Hypothermia, Acetylcholine, Body Temperature, Heart Arrest
Heart, Hypothermia, Acetylcholine, Body Temperature, Heart Arrest
citations This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 17 | |
popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Average | |
influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 10% | |
impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Top 10% |