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https://doi.org/10.31219/osf.i...
Article . 2023 . Peer-reviewed
License: CC BY
Data sources: Crossref
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Control of BST2 by NOD2.

descriptionPublicationkeyboard_double_arrow_right Article , Preprint 16 Sep 2023Publisher:Center for Open Science
Authors: Shahan Mamoor;

doi: 10.31219/osf.io/a69cb

Control of BST2 by NOD2.

Abstract

We provide evidence here from loss-of-function studies in mice and gain-of-function studies in human cells (1, 2) which demonstrate control of bone marrow stromal cell antigen 2, BST2, by the NLR (nucleotide-binding oligomerization domain-like receptor) family protein NOD2.

Keywords

Cell and Developmental Biology, Allergy and Immunology, Systems Biology, Immunity, Medicine and Health Sciences, Medical Specialties, Gastroenterology, Life Sciences, Genetics and Genomics, Genomics, Cell Biology, Immunology and Infectious Disease

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
0
Average
Average
Average
hybrid