
Most molecular targets for treating cancer prove to be busts. Broadly applicable anticancer therapies have been particularly difficult to develop because of the multiplicity of genetic mutations in a cancer cell: target one mutation and another will slip by, potentially causing the therapy to be
STAT3 Transcription Factor, Lymphoma, Non-Hodgkin, Antineoplastic Agents, Apoptosis, Breast Neoplasms, Oligonucleotides, Antisense, DNA-Binding Proteins, Mice, Neoplasms, Trans-Activators, Tumor Cells, Cultured, Animals, Humans
STAT3 Transcription Factor, Lymphoma, Non-Hodgkin, Antineoplastic Agents, Apoptosis, Breast Neoplasms, Oligonucleotides, Antisense, DNA-Binding Proteins, Mice, Neoplasms, Trans-Activators, Tumor Cells, Cultured, Animals, Humans
| selected citations These citations are derived from selected sources. This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 2 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Average | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 10% | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Average |
