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PLoS Biology
Article . 2023 . Peer-reviewed
License: CC BY
Data sources: Crossref
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PLoS Biology
Article . 2023
Data sources: Europe PubMed Central
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PubMed Central
Other literature type . 2023
License: http://creativecommons.org/licenses/by/4.0/This is an open access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/) , which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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PLoS Biology
Article . 2023
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Cell senescence, the senescence-associated secretory phenotype, and cancers

descriptionPublicationkeyboard_double_arrow_right Article , Other literature type 21 Sep 2023 English Publisher:Public Library of Science (PLoS)Journal:PLOS Biology, volume 21, page e3002326 (eissn: 1545-7885, Copyright policy )Funded by:NIH | Effect of Aging on Preadi..., NIH | Translational Geroscience..., NIH | Postdoctoral Training Pro...
Authors: Larissa G. P. Langhi Prata; Tamar Tchkonia; James L. Kirkland;

doi: 10.1371/journal.pbio.3002326

pmid: 37733806

pmc: PMC10547493

Cell senescence, the senescence-associated secretory phenotype, and cancers

Abstract

Cellular senescence is a cell fate caused by multiple stresses. A 2008 article in PLOS Biology reported a senescence-associated secretory phenotype that can promote inflammation and cancer, eventually enabling the development of senolytic drugs.

Related Organizations
Keywords

Inflammation, Phenotype, QH301-705.5, Neoplasms, Perspective, Humans, Cell Differentiation, Senescence-Associated Secretory Phenotype, Biology (General), Cellular Senescence

  • BIP!
    Impact byBIP!
    selected citations
    These citations are derived from selected sources.
    This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
    		 15
    popularity
    This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
    		 Top 10%
    influence
    This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
    		 Average
    impulse
    This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
    		 Top 10%
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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
15
Top 10%
Average
Top 10%
Green
gold
Related to Research communities
Knowmad Institut
Cancer Research