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Journal of Innate Immunity
Article . 2023 . Peer-reviewed
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Journal of Innate Immunity
Article . 2023
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Conference object . 2023
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Journal of Innate Immunity
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N6-Methyladenosine (m6A) Modification in Natural Immune Cell-Mediated Inflammatory Diseases

descriptionPublicationkeyboard_double_arrow_right Article , Conference object 01 Jan 2023 English Publisher:S. Karger AGJournal:Journal of Innate Immunity, volume 15, pages 804-821 (issn: 1662-811X, eissn: 1662-8128, Copyright policy )
Authors: Yan Teng; Jin Yi; Junnian Chen; Lu Yang;

doi: 10.1159/000534162

pmid: 37903470

pmc: PMC10673353

N6-Methyladenosine (m6A) Modification in Natural Immune Cell-Mediated Inflammatory Diseases

Abstract

The post-transcriptional N6-methyladenosine (m6A) modification of RNA influences stability, transport, and translation with implications for various physiological and pathological processes. Immune cell development, differentiation, and activation are also thought to be regulated by m6A and affect host defense against pathogens and inflammatory response with impacts on infectious, neoplastic, autoimmune, cardiovascular, hepatic, and osteal diseases. The current review summarizes recent research on m6A in monocyte/macrophages, neutrophils, dendritic cells, natural killer cells, and microglia and gives insights into epigenetic modifications of the immune system and novel therapeutic strategies for immune-related diseases.

Related Organizations
Keywords

Killer Cells, Natural, Neutrophils, R, Leukocytes, Medicine, Cell Differentiation, m6a, innate immune cell, Review Article, Internal medicine, RC31-1245, inflammatory disease

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    selected citations
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    This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
    		 10
    popularity
    This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
    		 Top 10%
    influence
    This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
    		 Average
    impulse
    This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
    		 Top 10%
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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
10
Top 10%
Average
Top 10%
Green
gold