Maternal diabetes induces senescence and neural tube defects sensitive to the senomorphic rapamycin
Copyright policy )NIH| The Role of C-Kit Positive Cardiac Progenitors in Maternal Diabetes-Induced Heart Defects and the Therapeutic Values of These Cells ,
NIH| Molecular Signalling Pathways and Cellular Stress in Diabetic Embryopathy ,
NIH| Epitranscriptomic Alteration and Planar Cell Polarity Signaling In Diabetic Embroyopathy ,
NIH| Molecular Signalling Pathways and Cellular Stress in Diabetic Embryopathy ,
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NIH| Apoptotic Mechanism of Maternal Diabetes-Induced Neural Tube Defects ,
NIH| Autophagy and its Regulation in Diabetic Embryopathy ,
NIH| MicroRNA-Suppressed Mitochondrial Fusion in Mediating the Teratogenicity of Maternal Diabetes Leading to Heart Defects
Cheng Xu; Wei-Bin Shen; E. Albert Reece; Hidetoshi Hasuwa; Christopher Harman; Sunjay Kaushal; Peixin Yang;
Maternal diabetes induces senescence and neural tube defects sensitive to the senomorphic rapamycin
A microRNA-activated pathway in maternal diabetes–induced neuroepithelial cell senescence leads to neural tube defects.
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These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
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This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
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NIH| The Role of C-Kit Positive Cardiac Progenitors in Maternal Diabetes-Induced Heart Defects and the Therapeutic Values of These Cells, NIH| Molecular Signalling Pathways and Cellular Stress in Diabetic Embryopathy, NIH| Epitranscriptomic Alteration and Planar Cell Polarity Signaling In Diabetic Embroyopathy, NIH| Molecular Signalling Pathways and Cellular Stress in Diabetic Embryopathy