Glomerular galactose‐deficient IgA1 detected in donor‐derived and recurrent IgA nephropathy

descriptionPublicationkeyboard_double_arrow_right Article 08 Sep 2021 English Publisher:Frontiers Media SAJournal:Transplant International, volume 34, pages 2,418-2,421 (issn: 0934-0874, eissn: 1432-2277,

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Authors: Su, Xuesong; Kim, Youngki; Riad, Samy; Spong, Richard; Bu, Lihong;

doi: 10.1111/tri.14009

pmid: 34378256

Glomerular galactose‐deficient IgA1 detected in donor‐derived and recurrent IgA nephropathy

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Abstract

Galactose deficient IgA1 (Gd-IgA1) plays an important role in the pathogenesis of IgA nephropathy (IgAN)1 . Immunofluorescence (IF) staining of KM55, a Gd-IgA1 specific monoclonal antibody, becomes an easy and reliable tool to detect Gd-IgA1 deposited in glomerular mesangium of IgAN.

Related Organizations

University of Minnesota
United States
China Medical University
China (People's Republic of)
University of Minnesota Morris
United States
First Hospital of China Medical University
China (People's Republic of)

Keywords

Transplantation, Kidney Glomerulus, Galactose, Humans, Glomerulonephritis, IGA, Tissue Donors, Immunoglobulin A

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