
We apologize to the many authors whose primary data could not be cited because of limitations of space. We thank Grace Watson for great assistance in preparation of the manuscript. This work is supported by NIH grant HD 37283.
Chromosomes, Human, Pair 15, Models, Genetic, Ubiquitin-Protein Ligases, Long-Term Potentiation, SNRPN, Brain, Ligases, Mice, Angelman syndrome, Genetics, Animals, Humans, Genetics(clinical), Prader-Willi syndrome, Angelman Syndrome, Prader-Willi Syndrome, Ubiquitins
Chromosomes, Human, Pair 15, Models, Genetic, Ubiquitin-Protein Ligases, Long-Term Potentiation, SNRPN, Brain, Ligases, Mice, Angelman syndrome, Genetics, Animals, Humans, Genetics(clinical), Prader-Willi syndrome, Angelman Syndrome, Prader-Willi Syndrome, Ubiquitins
| selected citations These citations are derived from selected sources. This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 175 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Top 10% | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 1% | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Top 1% |
