
PATIENTS with undiagnosed cold-reactive protein (CRP) disease undergoing hypothermia are at high risk of major morbidity and mortality if they are not identified and treated before surgery.1 Chronic thromboembolic pulmonary hypertension may be cured by pulmonary thromboendarterectomy (PTE), for which deep hypothermia and circulatory arrest (DHCA) are recommended.2 Because of hypothermia, PTE is a challenging procedure in patients with sickle cell and CRP disease. Successful PTE in patients with sickle cell disease has been reported by Yung et al.3 A patient is described with a preoperative diagnosis of cold agglutinins (CA) in whom successful PTE was also performed.
Male, deep hypothermia, 330, Chronic thromboembolic pulmonary hypertension, Pulmonary, Endarterectomy, Cryoagglutinin, Middle Aged, Pulmonary Artery, Antiphospholipid Syndrome, Chronic Disease, Humans, pulmonary thromboendarterectomy, Anemia, Hemolytic, Autoimmune, Pulmonary Embolism
Male, deep hypothermia, 330, Chronic thromboembolic pulmonary hypertension, Pulmonary, Endarterectomy, Cryoagglutinin, Middle Aged, Pulmonary Artery, Antiphospholipid Syndrome, Chronic Disease, Humans, pulmonary thromboendarterectomy, Anemia, Hemolytic, Autoimmune, Pulmonary Embolism
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