
doi: 10.1038/nn.2853
The sustained metabolic activation of the brain's default-mode network is thought to render the system vulnerable to Alzheimer's disease. Recent results with transgenic mice support this view by linking neuronal activity to interstitial fluid amyloid-β levels and the development of amyloid-β plaques.
info:eu-repo/classification/ddc/570, Neurons: metabolism, Plaque, Amyloid: pathology, Amyloid beta-Peptides, Mice, Transgenic, Amyloid beta-Peptides: metabolism, Brain: pathology, Disease Models, Animal, Mice, Alzheimer Disease: pathology, Neurons: pathology, Animals, Alzheimer Disease: metabolism
info:eu-repo/classification/ddc/570, Neurons: metabolism, Plaque, Amyloid: pathology, Amyloid beta-Peptides, Mice, Transgenic, Amyloid beta-Peptides: metabolism, Brain: pathology, Disease Models, Animal, Mice, Alzheimer Disease: pathology, Neurons: pathology, Animals, Alzheimer Disease: metabolism
| selected citations These citations are derived from selected sources. This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 28 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Top 10% | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 10% | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Top 10% |
