To the Editor: The recent article by Sethi et al., ‘Membranoproliferative glomerulonephritis and C3 glomerulopathy: resolving the confusion’, made me revisit a 12-year-old publication. In ‘Apparent progression of acute glomerulonephritis to dense deposit disease’ we described an 8-year-old boy with hypocomplementemia and meningococcemia. The first kidney biopsy showed glomeruli with abundant deposits of C3 and scarce deposits of IgG. Typical subepithelial humps were observed ultrastucturally, with only occasional subendothelial and intramembranous deposits. Two years later, a new biopsy was again positive for C3, and intramembranous, electron dense ribbon-like change, typical of dense-deposit disease (DDD), was present along the capillary loops, with only occasional subepithelial humps. In view of the clinical history of our patient, and the typical findings of DDD in the second biopsy, I suggest that the first biopsy, with its scarce subendothelial deposits and no membranoproliferative pattern, might also represent C3 glomerulopathy, similar to cases described by Fakhouri et al. and Servais et al. One might propose that our case illustrates that C3 glomerulopathy can present with variable faces in the same patient.