
Neovascularisation is a major cause of visual loss in a number of ophthalmic diseases. This review aims to outline the basic regulators of vessel growth in corneal neovascularisation. An understanding of the underlying principles of physiological and pathophysiological vascular development helps to appreciate current approaches to prevent or treat corneal neovascularisation. Options for future interventions will be discussed in the light of recent evidence provided by animal models of corneal neovascularisation.
Bevacizumab, Vascular Endothelial Growth Factor A, Disease Models, Animal, Mice, Animals, Humans, Angiogenesis Inhibitors, Corneal Neovascularization, Genetic Therapy, Antibodies, Monoclonal, Humanized
Bevacizumab, Vascular Endothelial Growth Factor A, Disease Models, Animal, Mice, Animals, Humans, Angiogenesis Inhibitors, Corneal Neovascularization, Genetic Therapy, Antibodies, Monoclonal, Humanized
| selected citations These citations are derived from selected sources. This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 36 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Top 10% | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Top 10% | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Average |
