THE short survival of erythrocytes given by transfusion with stored donor blood is a problem of great current interest. The well-known steadily decreasing intensity of glycolysis in stored blood, despite excess of glucose, is easily explainable by a loss of adenosine triphosphate1. Gabrio et al. 1–3 have shown that addition of adenosine or inosine re-establishes the cell content of organic phosphate, increases the consumption of glucose, and thus the post-transfusion viability.