
pmid: 27886894
Homonymous hemianopia from stroke causes visual disability. Although some patients experience spontaneous improvement, others have limited to no change and may be left with a severe disability. Current rehabilitation strategies are compensatory and cannot restore function. Animal studies suggest that central nervous system plasticity could allow for redirection of lost visual function into undamaged areas of cortex. A commercial therapy system was developed, from which claims of visual field expansion were disputed by independent researchers. The treatment remains controversial with seemingly contradictory data being generated. Continued research is underway to demonstrate the (non-)efficacy of this treatment method.
Stroke, Neuronal Plasticity, Hemianopsia, Humans, Recovery of Function, Visual Fields
Stroke, Neuronal Plasticity, Hemianopsia, Humans, Recovery of Function, Visual Fields
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