
There is now considerable evidence from both experimental and clinical studies that immune and inflammatory processes can contribute to the onset of stroke and the neurologic and psychologic outcomes. Several specific therapeutic targets have been identified that may significantly improve the devastating impact of stroke.
Neurons, Cell Death, Free Radicals, Sick Role, Brain, Psychoneuroimmunology, Cerebral Infarction, Stroke, Animals, Cytokines, Humans, Brain Damage, Chronic, Microglia, Chemokines, Encephalomyelitis, Cerebral Hemorrhage
Neurons, Cell Death, Free Radicals, Sick Role, Brain, Psychoneuroimmunology, Cerebral Infarction, Stroke, Animals, Cytokines, Humans, Brain Damage, Chronic, Microglia, Chemokines, Encephalomyelitis, Cerebral Hemorrhage
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| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Average | |
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