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Molecular Cell
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Molecular Cell
Article . 2016 . Peer-reviewed
License: Elsevier Non-Commercial
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Article . 2017
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https://dx.doi.org/10.1016/j.m...
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Defective Autophagy Initiates Malignant Transformation

descriptionPublicationkeyboard_double_arrow_right Article 01 May 2016 English Publisher:Elsevier BVJournal:Molecular Cell, volume 62, pages 473-474 (issn: 1097-2765, Copyright policy )
Authors: Lorenzo, Galluzzi; José Manuel, Bravo-San Pedro; Guido, Kroemer;

doi: 10.1016/j.molcel.2016.05.001

pmid: 27203173

Defective Autophagy Initiates Malignant Transformation

Abstract

In this issue of Molecular Cell, Park et al. (2016) elegantly demonstrate that a partial defect in autophagy supports malignant transformation as it favors the production of genotoxic reactive oxygen species by mitochondria.

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Keywords

Cell Transformation, Neoplastic, Autophagy, Reactive Oxygen Species, DNA Damage, Mitochondria

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    selected citations
    These citations are derived from selected sources.
    This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
    		 22
    popularity
    This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
    		 Top 10%
    influence
    This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
    		 Average
    impulse
    This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
    		 Top 10%
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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
22
Top 10%
Average
Top 10%
hybrid
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