Chromatin Modifiers, Cognitive Disorders, and Imprinted Genes
Copyright policy )Chromatin Modifiers, Cognitive Disorders, and Imprinted Genes
In this issue of Developmental Cell, Kernohan et al. link the chromatin regulatory proteins ATRX, MeCP2, CTCF, and cohesin with silencing of H19 and other imprinted genes during critical stages of postnatal brain development, perhaps suggesting a common etiology for several human diseases that exhibit defects in brain development and function.
- National Institute of Child Health and Human Development United States
- National Institute of Health Pakistan
- National Institutes of Health United States
Male, CCCTC-Binding Factor, RNA, Untranslated, Chromosomal Proteins, Non-Histone, Methyl-CpG-Binding Protein 2, DNA Helicases, Brain, Nuclear Proteins, Cell Cycle Proteins, Chromatin, Repressor Proteins, Genomic Imprinting, Mice, Insulin-Like Growth Factor II, Animals, Humans, Female, RNA, Long Noncoding, Gene Silencing, Cognition Disorders, Developmental Biology
Male, CCCTC-Binding Factor, RNA, Untranslated, Chromosomal Proteins, Non-Histone, Methyl-CpG-Binding Protein 2, DNA Helicases, Brain, Nuclear Proteins, Cell Cycle Proteins, Chromatin, Repressor Proteins, Genomic Imprinting, Mice, Insulin-Like Growth Factor II, Animals, Humans, Female, RNA, Long Noncoding, Gene Silencing, Cognition Disorders, Developmental Biology
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