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Cell Reports
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Cell Reports
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Mechanism of Nonsense-Mediated mRNA Decay Stimulation by Splicing Factor SRSF1

Authors: Aznarez, Isabel; Nomakuchi, Tomoki T.; Tetenbaum-Novatt, Jaclyn; Rahman, Mohammad Alinoor; Fregoso, Oliver; Rees, Holly; Krainer, Adrian R.;

Mechanism of Nonsense-Mediated mRNA Decay Stimulation by Splicing Factor SRSF1

Abstract

The splicing factor SRSF1 promotes nonsense-mediated mRNA decay (NMD), a quality control mechanism that degrades mRNAs with premature termination codons (PTCs). Here we show that transcript-bound SRSF1 increases the binding of NMD factor UPF1 to mRNAs while in, or associated with, the nucleus, bypassing UPF2 recruitment and promoting NMD. SRSF1 promotes NMD when positioned downstream of a PTC, which resembles the mode of action of exon junction complex (EJC) and NMD factors. Moreover, splicing and/or EJC deposition increase the effect of SRSF1 on NMD. Lastly, SRSF1 enhances NMD of PTC-containing endogenous transcripts that result from various events. Our findings reveal an alternative mechanism for UPF1 recruitment, uncovering an additional connection between splicing and NMD. SRSF1's role in the mRNA's journey from splicing to decay has broad implications for gene expression regulation and genetic diseases.

Keywords

Cell Nucleus, Models, Genetic, Serine-Arginine Splicing Factors, Amino Acid Motifs, RNA-Binding Proteins, Exons, Article, Nonsense Mediated mRNA Decay, Alternative Splicing, Codon, Nonsense, Humans, RNA, Messenger, Phosphorylation, HeLa Cells, Protein Binding, Transcription Factors

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
65
Top 1%
Top 10%
Top 10%
Green
gold