
pmid: 28388403
Necroptosis is a highly inflammatory form of programmed cell death that results from MLKL-mediated disruption of the cell membrane. In this issue of Cell, Gong et al. challenge the notion that MLKL activation is a point of no return by identifying mechanisms to counterbalance necroptosis, sustain plasma membrane integrity, and prolong cell viability.
Necrosis, Receptor-Interacting Protein Serine-Threonine Kinases, Apoptosis, Phosphorylation, Protein Kinases
Necrosis, Receptor-Interacting Protein Serine-Threonine Kinases, Apoptosis, Phosphorylation, Protein Kinases
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