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IDH1 and IDH2: Not Your Typical Oncogenes

descriptionPublicationkeyboard_double_arrow_right Article 01 Mar 2010 English Publisher:Elsevier BVJournal:Cancer Cell, volume 17, pages 215-216 (issn: 1535-6108, Copyright policy )
Authors: Reitman, Zachary J.; Parsons, D. Williams; Yan, Hai;

doi: 10.1016/j.ccr.2010.02.024

pmid: 20227034

pmc: PMC4467912

IDH1 and IDH2: Not Your Typical Oncogenes

Abstract

Oncogenes usually increase their normal function when activated. However, seemingly oncogenic mutations in IDH1 and IDH2 reduce their native enzyme activity. In this issue of Cancer Cell, Ward et al. pin down a neomorphic enzyme activity as a possible oncogenic function for these alterations.

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Keywords

Cancer Research, Oncology, Cell Biology

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    This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
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    popularity
    This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
    		 Top 10%
    influence
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    impulse
    This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
63
Top 10%
Top 10%
Top 10%
hybrid
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Cancer Research