descriptionPublicationkeyboard_double_arrow_right Article 01 Feb 2023 English Publisher:Elsevier BVJournal:Autoimmunity Reviews, volume 22, page 103,241 (issn: 1568-9972,

Authors: Okrój, Marcin; Rodríguez de Córdoba, Santiago;

doi: 10.1016/j.autrev.2022.103241

pmid: 36494043

handle: 10261/287925

Will a hyperactive classical complement pathway exacerbate autoimmune diseases?

- Summary
- Subjects
- Metrics

Abstract

No specific funding was obtained for this study. SRdC is supported by Comunidad de Madrid, Grant Numbers S2017/BMD-3673 and S2022/ BMD-7278, and Ministerio de Economía y Competitividad, Grant Number PID2019-104912RB-I00.

Peer reviewed

3 p.-1 tab.

Related Organizations

Keywords

Complement system, autoimmunity, genomic analyses, Autoimmunity, Complement System Proteins, Genomic analyses, Autoimmune Diseases, Systemic lupus erythematosus, systemic lupus erythematosus, DAMP, Humans, Lupus Erythematosus, Systemic, Complement Pathway, Classical, Complement Activation, complement system

Impact byBIP!

	citations This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).	0
	popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.	Average
	influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).	Average
	impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.	Average