The effect of camphor on mitochondrial respiration

descriptionPublicationkeyboard_double_arrow_right Article 01 Feb 1982 English Publisher:Springer Science and Business Media LLCJournal:Experientia, volume 38, pages 236-237 (issn: 0014-4754, eissn: 1420-9071,

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Authors: D F, Guilland-Cumming; G J, Smith;

doi: 10.1007/bf01945085

pmid: 7060709

The effect of camphor on mitochondrial respiration

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Abstract

Camphor at less than 8 mumoles/mg protein reduced the rate of oxygen consumption by rat liver mitochondria. The effect occurs only with NAD+-linked substrates. Succinate linked respiration was inhibited but this appears to be caused by some conversion of succinate to malate. At higher levels, camphor increases oxygen consumption with succinate substrate, by uncoupling at site II.

Keywords

Male, Succinic Acid, Mitochondria, Liver, Rats, Inbred Strains, Succinates, NAD, Camphor, Rats, Oxygen Consumption, Rotenone, Animals

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