
AbstractTwo major classes of herbicides include inhibitors of protoporphyrinogen oxidase (PPO) and phytoene desaturase (PDS). Plants can evolve resistance to PPO and PDS inhibitors via several mechanisms that include physical changes, resulting in reduced uptake, physiological changes, resulting in compartmentalization or altered translocation, and biochemical changes, resulting in enhanced metabolic degradation or alterations of protein structures, leading to loss of sensitivity to the herbicides. This review discusses the involvement of some of these mechanisms in the various cases of resistance to PDS‐ and PPO‐inhibiting herbicides, and highlights unique aspects of target‐site resistance to these herbicides. © 2014 Society of Chemical Industry
chlorophylls, codon usage, Herbicides, dual targeting, carotenoids, Plant Weeds, codon deletion, protoporphyrinogen oxidase, phytoene desaturase, invasive weed, aquatic weed management, herbicide resistance, somatic mutation, Protoporphyrinogen Oxidase, Enzyme Inhibitors, Oxidoreductases, Herbicide Resistance
chlorophylls, codon usage, Herbicides, dual targeting, carotenoids, Plant Weeds, codon deletion, protoporphyrinogen oxidase, phytoene desaturase, invasive weed, aquatic weed management, herbicide resistance, somatic mutation, Protoporphyrinogen Oxidase, Enzyme Inhibitors, Oxidoreductases, Herbicide Resistance
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