Paclitaxel‐induced ST‐Segment Elevations

descriptionPublicationkeyboard_double_arrow_right Article 20 Apr 2009 English Publisher:WileyJournal:Clinical Cardiology, volume 32 (issn: 0160-9289, eissn: 1932-8737,

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Authors: Gökmen, Gemici; Altuğ, Cinçin; Muzaffer, Değertekin; Ahmet, Oktay;

doi: 10.1002/clc.20291

pmid: 19382279

pmc: PMC6653473

Paclitaxel‐induced ST‐Segment Elevations

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Abstract

AbstractA 51‐year‐old woman presented with severe chest pain minutes after starting intravenous paclitaxel as a part of the systemic chemotherapy due to ovarian carcinoma. The electrocardiogram (ECG) revealed sinus rhythm with ST‐segment elevations in inferior and anterior leads. The ST‐segment elevations resolved immediately after sublingual nitroglycerine. Cardiac troponin T and CPK MB levels remained in the normal range at repeat measurements. It was presumed that in spite of standard premedication, paclitaxel had induced acute coronary syndrome with ST‐segment elevations in this patient. Copyright © 2009 Wiley Periodicals, Inc.

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Keywords

Ovarian Neoplasms, Paclitaxel, Carcinoma, Administration, Sublingual, Coronary Vasospasm, Middle Aged, Coronary Angiography, Antineoplastic Agents, Phytogenic, Angina Pectoris, Electrocardiography, Nitroglycerin, Treatment Outcome, Creatine Kinase, MB Form, Humans, Female, Stents, Acute Coronary Syndrome, Angioplasty, Balloon, Coronary, Infusions, Intravenous, Biomarkers

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