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Участие пуринорецепторов и инфекционной патологии в нарушениях сократительной деятельности матки во время беременности

Участие пуринорецепторов и инфекционной патологии в нарушениях сократительной деятельности матки во время беременности

Abstract

Presented was the current data on the role of adrenergic and purinergic nervous system in the regulation of labor activity, as well as inflammatory processes in the fallopian tubes that could change their anatomical and histological structure, the function of the receptor apparatus which can lead to disturbances of uterine contractile activity.

Изложены современные данные о роли адренергической и пунергической нервной системы в регуляции родовой деятельности, а также воспалительных процессов в области маточных труб, способных изменять их анатомо-гистологическое строение, состояние рецепторного аппарата и в результате приводить к нарушениям их сократительной активности.

Keywords

сократительная деятельность матки, ƒ-адренорецепторы, пуринергическая нервная система, р2-рецепторы, аденозин, внеклеточная аденозин-5'-трифосфорная кислота (атф), инфекционновоспалительные заболевания, кампилобактер, ƒ-adrenergic receptors, extracellular adenosine 5'-triphospate (atp)

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selected citations
These citations are derived from selected sources.
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Citations provided by BIP!
popularity
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
BIP!Popularity provided by BIP!
influence
This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
BIP!Influence provided by BIP!
impulse
This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.
BIP!Impulse provided by BIP!
0
Average
Average
Average
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