In recent years, the symptom of akinesia in parkinsonism has provided a valuable window into the physiological substrate of voluntary movement in man, particularly the role of the basal ganglia and the influence of dopamine. In a similar way, the study of individual nonmotor symptoms in parkinsonism can provide insights into the subsystems underlying discrete aspects of human cognition, emotion, and motivation. Even more importantly perhaps, their study in parkinsonism encourages us to examine the ways in which such diverse functions are integrated and influence broader categories of action and behavior (see Chapter 8 in the volume). In doing so, we are led to question whether the term “movement disorder” is the most appropriate term to apply to diseases such as parkinsonism.