mir-210: A Sensor for Hypoxic Stress during Tumorigenesis
Copyright policy )mir-210: A Sensor for Hypoxic Stress during Tumorigenesis
In this issue of Molecular Cell, Huang et al. (2009) demonstrate that hypoxia-inducible mir-210 acts as a rheostat for cellular adaptation and survival by inhibiting tumor initiation.
- Abramson Cancer Center United States
- University of Pennsylvania United States
- Howard Hughes Medical Institute United States
Homeodomain Proteins, Transcription, Genetic, Receptor, Fibroblast Growth Factor, Type 5, Cell Biology, Genetic Therapy, Hypoxia-Inducible Factor 1, alpha Subunit, Cell Hypoxia, Up-Regulation, Gene Expression Regulation, Neoplastic, MicroRNAs, Cell Transformation, Neoplastic, Stress, Physiological, Neoplasms, Animals, Humans, Gene Silencing, Homeobox A1 Protein, Molecular Biology, Cell Proliferation, Protein Binding, Transcription Factors
Homeodomain Proteins, Transcription, Genetic, Receptor, Fibroblast Growth Factor, Type 5, Cell Biology, Genetic Therapy, Hypoxia-Inducible Factor 1, alpha Subunit, Cell Hypoxia, Up-Regulation, Gene Expression Regulation, Neoplastic, MicroRNAs, Cell Transformation, Neoplastic, Stress, Physiological, Neoplasms, Animals, Humans, Gene Silencing, Homeobox A1 Protein, Molecular Biology, Cell Proliferation, Protein Binding, Transcription Factors
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