
pmid: 19942
Studies of mitochondrial function after circulatory shock, hemorrhagic or endotoxic, have revealed defective oxidative metabolism in several organs, such as the liver, kidney, and the brain (1,2,3). In this comment I would like to emphasize certain important aspects of these findings.
Shock, Hydrogen-Ion Concentration, Shock, Hemorrhagic, Shock, Septic, Mitochondria, Oxygen Consumption, Ischemia, Animals, Calcium, Gerbillinae
Shock, Hydrogen-Ion Concentration, Shock, Hemorrhagic, Shock, Septic, Mitochondria, Oxygen Consumption, Ischemia, Animals, Calcium, Gerbillinae
| citations This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | 4 | |
| popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network. | Average | |
| influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically). | Average | |
| impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network. | Average |
